Coconut Boosts Brain Function
The human brain needs a continual supply of fuel. When this continual supply is interrupted brain cells begin to die. In the short-term, this can cause symptoms such as headaches, brain fog, & tiredness. Long-term exposure to environments that deplete sources of brain fuel from effectively getting into the cells leads to neurodegenerative diseases. New research is showing that coconut oil can supply the brain with a very clean source of energy that prevents unwanted short-term symptoms and is effective at preventing and treating neurodegenerative disease states (1).
In many neurodegenerative conditions such as Alzheimer’s, neurons in certain regions of the brain have become insulin resistant. Insulin is the necessary hormone that brings glucose into cells where it can be used for energy. This is Diabetes in the brain. When brain cells become resistant to insulin they are unable to get glucose and they become malnourished and begin to die. Because the brain is unable to lower circulating blood sugar levels these levels remain high.
High Blood Sugar and Brain Function:
High blood sugar is neurotoxic in that it binds with protein molecules to create advanced glycolytic enzymes (AGE’s) (2, 3, 4). These AGE’s dramatically increase oxidative stress levels in the body and have a particular affinity for damaging neuronal tissue (2). This combination of high blood sugar and low energy states within the brain cells is the perfect storm for massive brain cell death.
Coconut Oil is considered one of the best fuels for healthy brain function (5, 6). Outside of mother’s milk, coconut oil is nature’s richest source of medium chain trigylcerides (MCT’s). Medium chain triglycerides (MCT) are not processed by your body in the same manner as long chain triglycerides. Normal fat metabolism depends on bile salts that have been released from your
gallbladder before it can be broken down in your digestive system.
Coconut Creates Ketones to Fuel the Brain
MCT’s bypass bile metabolism and go directly to your liver where they are converted into ketones (7). The liver then immediately releases the ketones into the bloodstream where they are transported to the brain to be used as fuel. Research has shown that the ketone bodies produced by MCT’s provide a stable source of energy for the brain during periods of low blood sugar without the neurological risks associated with high blood sugar (8).
This is why ketones appear to be the preferred source of brain food in patients affected by diabetes or any neurodegenerative conditions such as Alzheimer’s, Parkinson’s, ALS, & Multiple Sclerosis, etc. (9). One of the leading authorities on MCT research is Dr. Mary Newport. She has shown that ketone bodies may help the brain recover from lack of oxygen induced brain death in newborns through adults. (10). Additionally, ketones can help the heart patient recover from a heart attack and they can affectively shrink cancerous tumors.
Coconut Boosts Brain Function:
Additional research has shown that regular MCT consumption in dogs with “age-related mental decline” increases the phospholipid and omega-3 fatty acid (EPA & DHA) content in the parietal cortex of the brain (11). Omega 3 fatty acids are found in fish not coconut oil. The researchers believe that the presence of the MCT’s allowed omega 3’s to be liberated from fat stores & utilized in the brain where they are most needed to help form solid memory centers.
The average sized adult should consume 3-4 tablespoons of coconut oil daily. This could be taken orally, added to food, or applied onto skin. It can be taken in the form of coconut oil, milk, meat, or flakes. Coconut milk provides approximately 1 Tbsp of coconut oil for every 3 ounces. People with diabetes (type 1 & II), or neurological disease would benefit from up to 8-10 tablespoons of coconut oil daily.
Sources For This Article Include:
- Nafar F, Mearow KM. Coconut oil attenuates the effects of amyloid-β on cortical neurons in vitro. J Alzheimers Dis. 2014;39(2):233-7. PMID: 24150106
- Giacco F, Brownlee M. Oxidative stress and diabetic complications. Circ Res. 2010 Oct 29;107(9):1058-70. PMID: 21030723
- Kikuchi S, Shinpo K, Takeuchi M, Yamagishi S, Makita Z, Sasaki N, Tashiro K. Glycation–a sweet tempter for neuronal death. Brain Res Brain Res Rev. 2003 Mar;41(2-3):306-23. PMID: 12663085
- Takeuchi M, Kikuchi S, Sasaki N, Suzuki T, Watai T, Iwaki M, Bucala R, Yamagishi S. Involvement of advanced glycation end-products (AGEs) in Alzheimer’s disease. Curr Alzheimer Res. 2004 Feb;1(1):39-46. PMID: 15975084
- Napoletan A. Can Coconut Oil Prevent Alzheimer’s? Memory Loss and Alzheimer’s Prevention. Alzheimers.net: Link Here
- McDonald TS, Tan KN, Hodson MP, Borges K. Alterations of hippocampal glucose metabolism by even versus uneven medium chain triglycerides. J Cereb Blood Flow Metab. 2014 Jan;34(1):153-60. PMID: 24169853
- St-Onge MP, Jones PJ. Physiological effects of medium-chain triglycerides: potential agents in the prevention of obesity. J Nutr. 2002 Mar;132(3):329-32. PMID: 11880549
- Page KA, Williamson A, Yu N, McNay EC, Dzuira J, McCrimmon RJ, Sherwin RS. Medium-chain fatty acids improve cognitive function in intensively treated type 1 diabetic patients and support in vitro synaptic transmission during acute hypoglycemia. Diabetes. 2009 May;58(5):1237-44. PMID: 19223595
- Stafstrom CE, Rho JM. The ketogenic diet as a treatment paradigm for diverse neurological disorders. Front Pharmacol. 2012 Apr 9;3:59. PMID: 22509165
- Newport M. Alzheimer’s Disease: What If There Was a Cure? Link Here
- Taha AY, Henderson ST, Burnham WM. Dietary enrichment with medium chain triglycerides (AC-1203) elevates polyunsaturated fatty acids in the parietal cortex of aged dogs: implications for treating age-related cognitive decline. Neurochem Res. 2009 Sep;34(9):1619-25. PMID: 19301124